Absence Of Cpla2 In Lrp1 Smooth Muscle Cell Deficient Mice Promotes Severe Aortic Atherosclerotic Disease
This activity was presented at 2020 VRIC as part of Abstract Session 2: Atherosclerotic Disease and Role of the Immune System
Results from our studies will identify molecular mechanisms in the process of underpinning novel therapeutic targets for the devastating consequences of treating atherosclerotic disease plaque vulnerability and rupture by studying bioactive lipid dysregulation.
Professional Practice Gap
Our studies aim to understand the molecular mechanisms of atherosclerotic plaque vulnerability and rupture that continue to cause life-threatening complications affecting millions of people around the world. Our studies aim to understand atherosclerosis disease development via the abrogation of the inflammatory process by altered activation of the bioactive lipid machinery.
To create a virtual environment for the exchange of basic and translational vascular science that stimulates thoughtful discussion and motivates participants to discover solutions to important problems affecting vascular patients.
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